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Volume 14 (1995)
- Issue 3 Pages 151-
- Issue 2 Pages 91-
- Issue 1 Pages 1-
Volume 14, Issue 3
Displaying 1-8 of 8 articles from this issue
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RYOU TSUCHIYA, HIROMICHI AKIZUKI, KEN-ICHI MICHI1995 Volume 14 Issue 3 Pages 151-155
Published: December 01, 1995
Released on J-STAGE: June 08, 2010
JOURNAL FREE ACCESSErythema Exsudativum Multiforme is an acute inflammatory lesion which appears on skin and mucous membrane as erythema, papulosum, blister and other various forms of skin eczema.
The patient was a 28-year-old male. Erythema, blisters and erosion were observed on the tongue, buccul mucosa, hard palate and upper pharynx. Blister and erosion were observed on lips and eyelids. Hemorrhage, pain and crust formation also existed. Blisters were obse-rved on palms and insteps. Sedes G® was administered just before these symptoms were observed. The symptoms disappeared with symptomatic treatment on the 11 th day after the patient's first visit to the clinic After the symptoms disappeared, reaction to medication was examined by conducting a patch test. The reaction was positive with 20 % solution of Sedes G® and 30 % Isopropylantipyrine. As the result of all the indications mentioned above, it has been concluded that this case should be Erythema Exsudativum Multiforme due to Sedes G®.View full abstractDownload PDF (5781K) -
TAKU SHIRAI, INTETSU KOBAYASHI1995 Volume 14 Issue 3 Pages 156-159
Published: December 01, 1995
Released on J-STAGE: June 08, 2010
JOURNAL FREE ACCESSFor prophylactic use of ceftriaxone intravenously, twenty blood samples after tooth extraction were cultured on Bactec NR 16 A® and NR 17 A® (Becton Dickinson, Maryland, U. S. A.) . Each MIC of ceftriaxone in identified microbes was measured. In addition the serum concentrations of ceftriaxone were measured. The incidence of bacteremia was 15 % (3 out of 20) and three strains were isolated. They were all anaerobes, and no streptococcal bacteremia were detected. The MICs of ceftriaxone were 0.39, 1.56 and 12.5 μg/ml. The serum levels varied from 95 to 277 μg/ml, which were satisfactory concentrations. The results suggested that intravenous ceftriaxone prophylaxis for tooth extraction decreased Bacteremia rates significantly, including the isolated rate of streptococci.View full abstractDownload PDF (328K) -
LEIF OLGART1995 Volume 14 Issue 3 Pages 160-164
Published: December 01, 1995
Released on J-STAGE: June 08, 2010
JOURNAL FREE ACCESSThe consideration of pulpal haemodynamics is naturally intermeshed with inflammatory responses. Cellular and humoral factors may be the vehicles which aid in physiologic regulation, but when these systems are overly activated, they may lead to pathologic changes. Sensory nerves may initiate inflammatory reactions when activated, and interestingly, recent findings indicate that vasoconstrictor nerves in the pulp can inhibit the release of nervously stored vasoactive and inflammatory mediators. Thus, there are mechanisms available for endogenous control of inflammation.
The ultimate goal for studies of the many components of inflammation in oral tissues is to find ways to interrupt or cure a harmful pathologic reaction. Since sensory nerves, e.g, in the dental pulp, are often the first structures to be activated during clinical procedures, reactions that eventually occur can be assumed to be initiated and perpetuated by the sensory neuropeptides. It is therefore probable that methods to reach the excitable structures by local administration of suitable drugs may some day be of clinical value. However, still a remaining obstacle, and a real challenge, is to find methods to clinically diagnose the state of health or disease of the encapsulated dental pulp.View full abstractDownload PDF (628K) -
ROBERT ORCHARDSON1995 Volume 14 Issue 3 Pages 165-170
Published: December 01, 1995
Released on J-STAGE: June 08, 2010
JOURNAL FREE ACCESSHypersensitive teeth are characterised by transient pains arising when mechanical, thermal, chemical or evaporative stimuli are applied to exposed dentine. The pain cannot be explained by dental defects or overt pulpal pathology, although the possibility of some inflammation in the pulps of hypersensitive teeth cannot be excluded. The clinical and morphological features can provide some clues about the aetiology of hypersensitive dentine. Current information suggests that hypersensitive dentine may be related to traumatic oral hygiene procedures and to factors such as regular consumption of acidic drinks. Dentine sensitivity may be assessed either in terms of the stimulus intensity necessary to elicit pain (sensory threshold) or as the subjectively rated degree of pain evoked by a standard stimulus. At present, many different methods of assessment are used in a variety of clinical trial designs, and this diversity of methodology makes it difficult to draw meaningful comparisons between different studies evaluating treatments for hypersensitive dentine. A greater degree of standardisation of methods is desirable in clinical trials to evaluate desensitising treatments.View full abstractDownload PDF (870K) -
1995 Volume 14 Issue 3 Pages 171-172
Published: December 01, 1995
Released on J-STAGE: June 08, 2010
JOURNAL FREE ACCESSDownload PDF (227K) -
Leif Olgart1995 Volume 14 Issue 3 Pages 180
Published: December 01, 1995
Released on J-STAGE: June 08, 2010
JOURNAL FREE ACCESSDownload PDF (98K) -
Robert Orchardson1995 Volume 14 Issue 3 Pages 181
Published: December 01, 1995
Released on J-STAGE: June 08, 2010
JOURNAL FREE ACCESSDownload PDF (128K) -
1995 Volume 14 Issue 3 Pages 182-221
Published: December 01, 1995
Released on J-STAGE: June 08, 2010
JOURNAL FREE ACCESSDownload PDF (5912K)
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