巻号一覧
14 巻 (1995)
- 3 号 p. 151-
- 2 号 p. 91-
- 1 号 p. 1-
14 巻, 3 号
選択された号の論文の8件中1～8を表示しています
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土屋 亮, 秋月 弘道, 道 健一1995 年 14 巻 3 号 p. 151-155
発行日: 1995/12/01
公開日: 2010/06/08
ジャーナル フリーErythema Exsudativum Multiforme is an acute inflammatory lesion which appears on skin and mucous membrane as erythema, papulosum, blister and other various forms of skin eczema.
The patient was a 28-year-old male. Erythema, blisters and erosion were observed on the tongue, buccul mucosa, hard palate and upper pharynx. Blister and erosion were observed on lips and eyelids. Hemorrhage, pain and crust formation also existed. Blisters were obse-rved on palms and insteps. Sedes G® was administered just before these symptoms were observed. The symptoms disappeared with symptomatic treatment on the 11 th day after the patient's first visit to the clinic After the symptoms disappeared, reaction to medication was examined by conducting a patch test. The reaction was positive with 20 % solution of Sedes G® and 30 % Isopropylantipyrine. As the result of all the indications mentioned above, it has been concluded that this case should be Erythema Exsudativum Multiforme due to Sedes G®.抄録全体を表示PDF形式でダウンロード (5781K) -
白井 卓, 小林 寅〓1995 年 14 巻 3 号 p. 156-159
発行日: 1995/12/01
公開日: 2010/06/08
ジャーナル フリー予防投与として, セフトリアキソン1gを点滴静注した際の, 抜歯後ならびに歯科観血処置後の菌血症の発現頻度をBactec NR 16 A, 17A (Becton Dickinson社, U.S.A.) を用いて検討した.20例中3例で菌陽性となり, その発現率は15%であった.検出菌3株は, いずれも嫌気性菌で, Streetococcusは1株も検出されなかった.3株に対するセフトリアキソンのMICは, 0.39, 1.56および12.5μg/ml, 同時に測定した血液培養施行時の血清中セフトリアキソン濃度は, 95～277μg/mlであった.セフトリアキソン1gの点滴予防投与により, 抜歯後の菌血症発現率は明らかに減少した.抄録全体を表示PDF形式でダウンロード (328K) -
オルガート リーフ1995 年 14 巻 3 号 p. 160-164
発行日: 1995/12/01
公開日: 2010/06/08
ジャーナル フリーThe consideration of pulpal haemodynamics is naturally intermeshed with inflammatory responses. Cellular and humoral factors may be the vehicles which aid in physiologic regulation, but when these systems are overly activated, they may lead to pathologic changes. Sensory nerves may initiate inflammatory reactions when activated, and interestingly, recent findings indicate that vasoconstrictor nerves in the pulp can inhibit the release of nervously stored vasoactive and inflammatory mediators. Thus, there are mechanisms available for endogenous control of inflammation.
The ultimate goal for studies of the many components of inflammation in oral tissues is to find ways to interrupt or cure a harmful pathologic reaction. Since sensory nerves, e.g, in the dental pulp, are often the first structures to be activated during clinical procedures, reactions that eventually occur can be assumed to be initiated and perpetuated by the sensory neuropeptides. It is therefore probable that methods to reach the excitable structures by local administration of suitable drugs may some day be of clinical value. However, still a remaining obstacle, and a real challenge, is to find methods to clinically diagnose the state of health or disease of the encapsulated dental pulp.抄録全体を表示PDF形式でダウンロード (628K) -
オーチャードソン ロバート1995 年 14 巻 3 号 p. 165-170
発行日: 1995/12/01
公開日: 2010/06/08
ジャーナル フリーHypersensitive teeth are characterised by transient pains arising when mechanical, thermal, chemical or evaporative stimuli are applied to exposed dentine. The pain cannot be explained by dental defects or overt pulpal pathology, although the possibility of some inflammation in the pulps of hypersensitive teeth cannot be excluded. The clinical and morphological features can provide some clues about the aetiology of hypersensitive dentine. Current information suggests that hypersensitive dentine may be related to traumatic oral hygiene procedures and to factors such as regular consumption of acidic drinks. Dentine sensitivity may be assessed either in terms of the stimulus intensity necessary to elicit pain (sensory threshold) or as the subjectively rated degree of pain evoked by a standard stimulus. At present, many different methods of assessment are used in a variety of clinical trial designs, and this diversity of methodology makes it difficult to draw meaningful comparisons between different studies evaluating treatments for hypersensitive dentine. A greater degree of standardisation of methods is desirable in clinical trials to evaluate desensitising treatments.抄録全体を表示PDF形式でダウンロード (870K) -
1995 年 14 巻 3 号 p. 171-172
発行日: 1995/12/01
公開日: 2010/06/08
ジャーナル フリーPDF形式でダウンロード (227K) -
Leif Olgart1995 年 14 巻 3 号 p. 180
発行日: 1995/12/01
公開日: 2010/06/08
ジャーナル フリーPDF形式でダウンロード (98K) -
Robert Orchardson1995 年 14 巻 3 号 p. 181
発行日: 1995/12/01
公開日: 2010/06/08
ジャーナル フリーPDF形式でダウンロード (128K) -
1995 年 14 巻 3 号 p. 182-221
発行日: 1995/12/01
公開日: 2010/06/08
ジャーナル フリーPDF形式でダウンロード (5912K)
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