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Volume 55 (2003)
- Issue 2-4 Pages 138-
Volume 55, Issue 2-4
Displaying 1-8 of 8 articles from this issue
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[in Japanese]2003 Volume 55 Issue 2-4 Pages 138-143
Published: December 30, 2003
Released on J-STAGE: February 19, 2013
JOURNAL FREE ACCESS -
Fumito Ito, Homei Suga, Yutaka Ogawa2003 Volume 55 Issue 2-4 Pages 144-150
Published: December 30, 2003
Released on J-STAGE: February 19, 2013
JOURNAL FREE ACCESSAmong the various methods of treating blepharoptosis, frontalis muscle suspension is selected for cases showing poor levator function. We attempted direct suspension using frontalis muscle flap, which does not require a donor site or artificial materials. Thus, donor site scar and foreign body reaction are eliminated. Frontalis muscle flap changes to levator aponeurosis-like tissue and maintains its dynamic action after surgery.View full abstractDownload PDF (3594K) -
Satoshi FUKUDA, Sayuri ONISHI, Yasuo NII, Kenji KUSUMOTO, Yutaka OGAWA2003 Volume 55 Issue 2-4 Pages 151-154
Published: December 30, 2003
Released on J-STAGE: February 19, 2013
JOURNAL FREE ACCESSTo reduce facial bone fracture correctly, fractured fragments must be released freely once and set to an original skeletal structure. At the same time, the dissection and the reduction technique should not make the closed vessels and nerves injured. Here a 47-year-old female case of massive arterial hemorrhage at reducing malar bone fracture is presented. In the pterigopalatine fossa, which is a narrow space, many important vessels and nerves, especially maxillary artery, passed. It is very difficult to control the hemorrhage from the injured maxillary artery, where is deep from facial surface and can be hardly managed from oral cavity. Preoperatively this case had had intermittent epistaxis and the opening mouth had been limited in 17 m m. Because the skeletal deformity of the fractured malar bone was mainly in the zygomatic arch, and the patient refuse d her facial skin incision, Gillies's temporal approach, which set the incision only at the temporal hairy area, was indicated. At reducing the malar bone correctly by this technique, massive arterial hemorrhage occurred in the nasal an d oral cavity. Although a gauze pressure hemostasis and Belloque tamponade were applied, the blood loss was up to 1,400 ml in the volume and could not be controlled at the time. Emergent angiography was per formed and the leakage of post-superior alveolar artery, which is a branch of maxillary artery, was detected as an injured sign. Then transcatheter embolization with coils was performed under radiographic monitoring and resulted in perfect hemostasis. The postoperative general condition was good and the normal function and the degree of opening mouth and the malar form were acquired. The experience that unexpected massive arterial hemorrhage at reducing a malar bone fracture was controlled with embolization procedure is reported.View full abstractDownload PDF (2686K) -
Hiroaki MATSUBARA, Hiroya MASAKI, Hiroshi KAMIHATA, Toshiji IWASAKA2003 Volume 55 Issue 2-4 Pages 155-161
Published: December 30, 2003
Released on J-STAGE: February 19, 2013
JOURNAL FREE ACCESSBone marrow cells contain progenitors for vascular endothelial cells (EC) and cardiomyocytes, and also release angiogenic factors (VEGF and bFGF). We have shown availability of implantation of bone marrow mononuclear cells (BMI) for patients with ischemic limbs (Lancet,2002; 360: 427-435). Here, we will talk about the effect of BMI on pig model with chronic ischemic myocardium and 1-year follow-up data for no-option patient (CCS-class-IV angina) treated with sole therapy by BMI. #1) Animal study: Chronic ischemia was prepared by ameroid constrictor in domestic pigs. NOGA electromechanical system was used to map hibernating regions and to inject BMc (30 sites,10 cells). Increases in systolic function (EF: 33 to 52%) and regional blood flow (2.1-fold), reduction of ischemic area (29% to 8%) were observed. Capillary numbers were markedly increased (3.6-fold) with incorporation of BMc, while trans-differentiation into cardiomyocytes was much less (-0.1% of viable myocytes). There was no bone formation (n=15). #2) Clinical Study: A 65-year-old man suffered fr om refractory angina (CCS class IV) in spite of bypass surgery (2 times) and angioplasty (5 times).0.05 ml of BMc was injected into hibernating focus identified by NOGA mapping (30 different sites,3 x 10 cells). Increase in EF (42 to 54%), reduction of ischemic area (16.5% to 3.8%), decrease in angina occurrence (Class IV to I) were observed 10 month after BMI. There was no substantial arrhythmia on 24 h Holter recordings performed monthly for 10 months. Blood supply to ischemic hibernating myocardium and myogenic cell grafting by cell therapy using BMI is a promising approach for prevention of myocardial remodeling.View full abstractDownload PDF (4711K) -
[in Japanese]2003 Volume 55 Issue 2-4 Pages 162
Published: December 30, 2003
Released on J-STAGE: February 19, 2013
JOURNAL FREE ACCESS -
[in Japanese]2003 Volume 55 Issue 2-4 Pages 163-166
Published: December 30, 2003
Released on J-STAGE: February 19, 2013
JOURNAL FREE ACCESS -
Yoshiya TAHASHI2003 Volume 55 Issue 2-4 Pages 167-174
Published: December 30, 2003
Released on J-STAGE: February 19, 2013
JOURNAL FREE ACCESS -
Seisuke TAKEMURA2003 Volume 55 Issue 2-4 Pages 175-180
Published: December 30, 2003
Released on J-STAGE: February 19, 2013
JOURNAL FREE ACCESS
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