The Japanese Journal of Veterinary Science
Online ISSN : 1881-1442
Print ISSN : 0021-5295
ISSN-L : 0021-5295
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Volume 33, Issue 2
Displaying 1-5 of 5 articles from this issue
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  • EXPERIMENTAL BORDETELLA BRONCHISEPTICA INFECTION IN GUINEA PIGS
    Masaro NAKAGAWA, Takeshi MUTO, Hachiharu YODA, Takeshi NAKANO, Kiyoshi ...
    1971 Volume 33 Issue 2 Pages 53-60
    Published: April 25, 1971
    Released on J-STAGE: February 13, 2008
    DOIhttps://doi.org/10.1292/jvms1939.33.53
    JOURNAL FREE ACCESS
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  • PRELIMINARY STUDIES ON DIAGNOSIS OF AVIAN INFECTIOUS BRONCHITIS BY FLUORESCENT ANTIBODY TECHNIQUE
    Yukio BITO, Takeshi HIRONAO, Taketsugu MASUI
    1971 Volume 33 Issue 2 Pages 61-67_1
    Published: April 25, 1971
    Released on J-STAGE: February 13, 2008
    DOIhttps://doi.org/10.1292/jvms1939.33.61
    JOURNAL FREE ACCESS
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    l. The growth of avian infectious bronchitis (IB) virus (IBV) in chicken kidneycell cultures was studied by the direct fluorescent antibody technique. The earliest visiblefluorescence was detected in a granularform in th cytoplasm 3 -4 hours after viral in-oculation. The fluorescence became diffuse gradually all over the cytoplasm, reaching amaximum at 12 hours.2. The occurrence of IBV antigen was studied on stamp-specimens prepared fromthe trachea of chickens inoculated with IBV. A detectable amount of fluorescence ap-peared 24 hours after virus inoculation. An almost constant level of fluorescence wasmaintained over an observation period of seven days. Fluorescence appeared in agranularform or diffusely in the cytoplasm.3. When a tracheal specimen from the normal chicken was stained with anti-IBfluorescent antibody, or when one from the IB-infected chicken was allowed to reactwith FITC-conjugated rnormal 7-globulin, no fluorescence was observed on the specimen.No fluorescence was detected from a tracheal specimen from the Newcastle-disease (ND)-infected chicken stained with anti-IB fluorescent antibody, though ND virus antigen wasobviously demonstrated with anti-ND virus fluorescent antibody. These findings indicatethat the fluorescence detected frorn the tracheal specimen from the IB-infected chickenstained with anti-[B fluorescent antibody reflects IBV-specific antigen.
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  • STUDIES ON THE CHOLINESTERASE OF THE CHICKEN : I. PSEUDO-CHOLINESTERASE OF THE PLASMA AND DIISOPROPYLFLUOROPHOSPHATE-RESISTANT PSEUDO-CHOLINESTERASE OF THE LIVER AND OTHER TISSUES
    Akira TANABE, Satoshi HORI, Tooru TORIUMI, Miyoshi IKEDA, Norimoto URA ...
    1971 Volume 33 Issue 2 Pages 69-79
    Published: April 25, 1971
    Released on J-STAGE: February 13, 2008
    DOIhttps://doi.org/10.1292/jvms1939.33.69
    JOURNAL FREE ACCESS
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    [in Japanese]
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  • STUDIES ON CYTOPATHOGENIC VIRUSES FROM CATS WITH RESPIRATORY INFECTIONS : II. CHARACTERIZATION ON FELINE PICORNAVIRUSES
    Eiji TAKAHASHI, Shin-ichiro KONISHI, Manabu OGATA
    1971 Volume 33 Issue 2 Pages 81-87
    Published: April 25, 1971
    Released on J-STAGE: February 13, 2008
    DOIhttps://doi.org/10.1292/jvms1939.33.81
    JOURNAL FREE ACCESS
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    [in Japanese]
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  • PATHOLOGICAL FEATURES OF OSMOSE POISONING IN A COW : NEUROPATHOLOGICAL OBSERVATION
    Hiroshi SATOH, Akira HASHIMOTO, Junji YAMAGIWA
    1971 Volume 33 Issue 2 Pages 89-105_8
    Published: April 25, 1971
    Released on J-STAGE: February 13, 2008
    DOIhttps://doi.org/10.1292/jvms1939.33.89
    JOURNAL FREE ACCESS
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    A one-year-old crossbred-Holstein which died, about 4 days after the onset of symp-torus, of osmose poisoning was histopathologically investigated from a general viewpoiratwith special consideration of neuropathology. A complementary experiment was carriedout in mice orally administered with osmose.Taking changes almost common to both cow and mice into consideration, the prin-cipal changes seen in the cow were classified as follows: (1) multiple hemorrhages ex-tended over the whole body, (2) microvascular alteration (in other words, edematousloosening and swelling of the walls of small blood vessels), (3) polyneuropathy, (4) cere-brat edema and degeneration of nerve cells in the central nervous system, (5) fattydegeneration of the liver and edema of the gall bladder, (6) destruction (karyorrhexis)of lymphocytes in the lymphoid organs, (7) ulceration in the forestomach and abomasum, and (8) decrease in number of cellular elements in the bone marrow.Chemical and histochemical examinations for detection of arsenic and fluorinefailed to give positive results in both cow and mice.The following pathological features were inferred as "predisposing changes" inthe morphological sense: (l) a considerable part of the process of nervous disturbances(polyneuropathy), (2) a considerable part of the process of microvascular alteration, and(3) a part of the ulceration in the forestomach and abomasum and a part of the myo-pathic process in the striated and smooth muscles.The idea "predisposing changes" may give a certain suggestion for the interpreta-tion of pathological features in naturally occurring cases of various types of poisoning.
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