Journal of the Japan Diabetes Society
Online ISSN : 1881-588X
Print ISSN : 0021-437X
ISSN-L : 0021-437X
Volume 46, Issue 6
Displaying 1-8 of 8 articles from this issue
  • Tele-Communication and Diabetes
    Toshihiro Utsugi, Hiroyuki Shimizu, Yoshio Ohyama, Masahiko Kurabayash ...
    2003 Volume 46 Issue 6 Pages 423-427
    Published: June 30, 2003
    Released on J-STAGE: March 02, 2011
    JOURNAL FREE ACCESS
    Education is a key feature of diabetes care. Diabetes education in rural and remote areas has been disadvantaged when accessing continuing education by diabetes specialists. To support patients with diabetes and medical staffs in remote areas, we have developed education using a video conferencing, a satellite network, and a cable television (CATV) network. Gunma University and a studio of Tsumagoi village or Ueno village, in the mountain area of Gunma prefecture, were connected with video conferencing, and the Tsumagoi studio and each residence of Tsumagoi and Ueno villages were connected by CATV through a satellite network. Programs were broadcast live and interactive. Patients and medical staffs in the Tsumagoi studio, asked questions of diabetes professionals, who answered via the TVscreen. Questionnaires showed this education to be useful, mainly because so many thus specialists were able to participate. Teleeducation thus facilitates communication between patients and medical staffs in remote area with professionals.
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  • Masahiro Tajika, Kunio Yamanouchi, Kiyo Sugimoto, Emi Suzuki, Arisa Ya ...
    2003 Volume 46 Issue 6 Pages 429-435
    Published: June 30, 2003
    Released on J-STAGE: March 02, 2011
    JOURNAL FREE ACCESS
    Diet therapy is a standard treatment for diabetes mellitus, but patients and their families find it difficult to cook the required food at home. The use of food delivery for diabetes treatment is spreading, so we evaluate the system for patients with diabetes mellitus. Twelve outpatients used the system for 3 months. Energy intake, HbA1c, total cholesterol, triglyceride, albumin, and quality of life (QOL) were measured before and after system use. QOL was evaluated by the Problem Areas in Diabetes (PAID) score. Energy intake before this study was 1841kcal/day. After three months of using the system, energy intake decreased significantly to 1640kcal/day (p<0.05). Based on the low energy intake, HbA1c improve from 7.2% to 7.0%(p=0.079) and triglyceride decreased significantly from 201.7 mg/dl to 158.8mg/dl (p<0.05). The PAID score did not change before and after this study. Food delivery is thus thought to be useful for patients with diabetes mellitus who can neither understand their own nutritional requirements nor cook a recommended diet property.
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  • [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    2003 Volume 46 Issue 6 Pages 437-440
    Published: June 30, 2003
    Released on J-STAGE: March 02, 2011
    JOURNAL FREE ACCESS
  • Yuko Akehi, Ikuyo Ogawa, Takashi Asano, Keizou Anzai, Reiko Nakagawach ...
    2003 Volume 46 Issue 6 Pages 441-446
    Published: June 30, 2003
    Released on J-STAGE: March 02, 2011
    JOURNAL FREE ACCESS
    We report findings for an autopsy case of a 38-year-old woman with Down's syndrome complicated by type 1 diabetes and Hashimoto's thyroiditis. Although the patient was first diagnosed with NIDDM based on her insulin secretion at age 16, her fasting C-peptide gradually decreased, necessitating insulin treatment at 20 years of age. Anti-GAD antibody, HLA DR 9 (DRB 1*0901) and thyroid antibodies were positive at 35 years of age. Due to diabetic complications progressing over the 20 years, she died of renal failure at age 38. Mitochondrial DNA 3357 (G-A) mutation and β3-adrenergic receptor gene polymorphism were also demonstrated in this case. A pathological examination showed the following: 1) histological findings for the thyroid were compatible with Hashimoto's thyroiditis. 2) The number of islets decreased markedly in the pancreatic tail with hardly any detectable βcells and, while αcells were relatively well preserved. These findings suggest that autoimmunity in addition to gene mutations may have caused her diabetes. An increased prevalence of type 1 diabetes in Down's syndrome is well established, but, its mechanism remains to be clarified. Future genetic studies are thus required to clarify the cause of type 1 diabetes associated with Down's syndrome.
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  • Sachiko Bandai, Masanori Iwase, Naoko Mukai, Udai Nakamura, Kojiro Ich ...
    2003 Volume 46 Issue 6 Pages 447-453
    Published: June 30, 2003
    Released on J-STAGE: March 02, 2011
    JOURNAL FREE ACCESS
    A 33 year old woman admitted for frequent vomiting when 10 weeks pregnant. She had developed diabetes mellitus at age 15 and was diagnosed as MODY. Pregnancy was detected under well-controlled conditions of 6.8% of HbA1c and normoalbuminuria. Hyperemesis gravidarum was diagnosed and parental hyperalimentation implemented. Although she had no thyrotoxic symptoms or struma, hyperthyrodism was detected (free T4 1.9ng/dl, free T3 4.5pg/ml, TSH<0.03μU/ml). Since antithyroid or TSH receptor antibodies were negative and serum chorionic gonadotropin (hCG) was 147, 600mU/ml, hyperthyroidism due to elevated serum hCG was diagnosed. Her symptoms and hyperthyroidism were ameliorated along with the lowering of serum hCG until 18 weeks of gestation. In the second term, however, she developed toxemia of pregnancy and was readmitted at 31 weeks of gestation. At 36 weeks of gestation, a normal fetus was delivered by Caesarean section. After delivery, diabetic retinopathy and hypertension were transiently aggravated and urinary albumin excretion did not normalize until one year after delivery. hCG may thus induce transient hyperthyroidism and hyperemesis gravidarum due to its thyroid stimulating activity and may be related to the development of preeclampsia.
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  • Effect of VLCD and LCD Therapy on Gonadal Function
    Atsushi Kita, Hironori Yamasaki, Kennichi Yamakawa, Shiori Saigo, Tets ...
    2003 Volume 46 Issue 6 Pages 455-461
    Published: June 30, 2003
    Released on J-STAGE: March 02, 2011
    JOURNAL FREE ACCESS
    Obesity is an important causative factor in inducing insulin resistance and affecting gonadal function in women. We studied an obese diabetic woman with amenorrhea and hyperandrogenism such as hypertrichosis and acanthosis nigricans. Very low calorie diet (VLCD) and LCD therapy to this patient resulted in a 21.5 kg weight loss. Visceral fat area analyzed by abdominal CT was redued 48% and insulin resistance improved after VLCD LCD therapy. Elevated testosterone and androstendione serum were normalized from 89.0 to 21.6ng/dl and 3.5 to 1.1ng/ml. As reflected by sexual hormone ameriolation, her hypertrichosis and acanthosis nigricans almost disappeared 7 months after VLCD LCD therapy. Disappearance of hyperandrogenism may be attributed to improved insulin resistance and hyperinsulinemia. VLCD LCD therapy may thus be effective in ameliorating gonadal dysfunction in obese patients with hyperandrogenism. Note that sex hormone abnormality could be complicated in severely obese patients, and that early treatment such as VLCD LCD therapy may be useful.
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  • [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    2003 Volume 46 Issue 6 Pages 463-467
    Published: June 30, 2003
    Released on J-STAGE: March 02, 2011
    JOURNAL FREE ACCESS
  • 2003 Volume 46 Issue 6 Pages 469-508
    Published: June 30, 2003
    Released on J-STAGE: March 02, 2011
    JOURNAL FREE ACCESS
    Download PDF (10228K)
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