All issues
Volume 67 (1991)
- Issue 12 Pages 1295-
- Issue 11 Pages 1231-
- Issue 10 Pages 1147-
- Issue 9 Pages 14-
- Issue 8 Pages 811-
- Issue 7 Pages 755-
- Issue 6 Pages 655-
- Issue 5 Pages 587-
- Issue 4 Pages 19-
- Issue 3 Pages 175-
- Issue 2 Pages 57-
- Issue 1 Pages 1-
- Issue Supplement-3 Pa･･･
- Issue Supplement-2 Pa･･･
- Issue Supplement-1 Pa･･･
Predecessor
Volume 27, Issue 4
Displaying 1-5 of 5 articles from this issue
- |<
- <
- 1
- >
- >|
-
Kota NAKAI1951 Volume 27 Issue 4 Pages 89-94_1,121
Published: July 20, 1951
Released on J-STAGE: September 24, 2012
JOURNAL FREE ACCESSIt has been supported by many authorities that pinealectomized rabbit invites the acceleration of growth and precocity of genitals, and they have believed that these deficiency syndromes after pinealectomy are caused only by the disappearance of this control hormone.
But Prof. Y. Tokumitsu has emphasized from may experiments that the deficiency syndromes of endocrine organs are not only caused by the lack of these hormones, but by the positive functional change of other endocrine organs.
The writer has studied the pathogenesis of the deficiency syndromes after pinealectomy and found the following results.
The infantile male mice injected 4-8 times every other day the serum of the pinealectomized rabbits, invite the acceleration of growth and precocity. Consequently there is an active substance which accelerates their growth and development of genitals in the serum of the pinealectomized rabbit. This active substance appears in the blood over 20 days after pinealectomy and is insoluble to organic solvent, and disappears by the removal of anterior pituitary. But this is not influenced by the extirpation of testicles, suprarenal bodies, thyroid or parathyroid gland. If three anterior pituitary bodies of normal infantile male rabbits are transplanted to normal male rabbit, the same active substance appears in his blood.
From the above mentioned experiments, we believe that the acceleration of growth and precocily after pinealectomy is caused not only by the disappearance of pineal hormone, but also by the remarkable quantitative increase of growth hormone ane gonadotropic hormone of anterior pituitary.View full abstractDownload PDF (2144K) -
Part I. Morphology of the ChestKamon AKASHI1951 Volume 27 Issue 4 Pages 95-100,121
Published: July 20, 1951
Released on J-STAGE: September 24, 2012
JOURNAL FREE ACCESSVital measurements were conducted of 114 ama (women divers) between the ages of 17 and 49 of Boshu Shirahama in Chiba Prefecture, and under the direction of Dr. Shiro Ogawara, the following eight quantitative measurements, seven indices and chest formulae were compiled and detailed statistical observations were made.
1) Measured items:
i) Chest-length ii) Front chest-length (Sternum length)
iii) Chest-depth I (Depth of upper chest)
iv) Chest-dephth II (Chest-depth)
v) Chest-depth III (Depth of lower chest)
vi) Chest-width A (Chest-width)
vii) Chest-width B (width of lower chest)
viii) Chest-width C (Width of rib-arch)
2) Indices:
i) Chest-length index (Front chest-length·100/Chest-length)
ii) Chest (length-depth) index I (Chest-depth I·100/Chest-length)
iii) Chest index II (Chest-depth II·100/Chest-length)
iv) Chest index III (Chest-depth III·100/Chest-length)
v) Chest-width index A (Chest-width B·100/Chest-width A)
vi) Chest-width index B (Chest-width C·100/Chest-width A)
vii) Chest-width index C (Chest-width C·100/Chest width B)
3) Chest formulae: Each index was classified into five or six groups according to its numerical quantity, and each group was marked from one to six. The chest length was designated as“L”, and the numerical formulae which were obtained as the result of arranging the makings in order: thusly, LI II III ABC, were called chest formulae.
4) Only chest index I and chest-width index C derived from the mean numbers of each index are found to be absolutely equivalent when compared with the results of Dr. Kato's observations of adult women residing in Tokyo.
All other indices indicated either that the ama were bigger or showed that tendency.
5) There remarkable differences in individual chest formulae, but the chest formula obtained from the mean numbers of each index is 434444; therefore, differing from Kato's formula of 3333333 for adult women in general.
6) In other words, the length of the sternum of the ama is longer compared to the length of the chest than adult women in general, and although there is no difference in thickness of the chest at the upper region, the thickness at the center of the sternum and the lower region of the chest are both thicker in comparison to the length of the chest.
Furthermore, in comparing the widths between the two types of women, it is noted that the ama is comparatively wider below the central region of the sternum.
These facts mean that the thickness and width of the ama are better developed than adult women in general.
7) By thus being able to simplify the morphology of the chest by numerical formulae through the determing of chest formulae, the author believes that there is a very deep significance in constitutional anthropological and morphological research.View full abstractDownload PDF (1124K) -
Part II. Morphology of the Lower TorsoKamon AKASHI1951 Volume 27 Issue 4 Pages 101-106,123
Published: July 20, 1951
Released on J-STAGE: September 24, 2012
JOURNAL FREE ACCESSVital measurements were conducted of 114 ama between the ages of 17 and 49 in Boshu Shirahama of Chiba prefecture, and under the direction of Dr. Shiro Ogawara, the following five quantitative measurements, seven indices and formulae for the lower torso were compiled and detailed statistical observations were made.
1) Measured items:
i) Length of front abdomen ii) Width of rib-arch iii) Pelvis width iv) Maximum width of thighs v) Minimum circumference of abdomen
2) Indices:
i) Lower-torso index I (Width of rib-arch·100/Length of front abdomen)
ii) Lower-torso index II (Pelvis width·100/Length of front abdomen)
iii) Lower-torso index III (Maximum width of thighs·100/Length of front abdomen)
iv) Lower-torso width index A (Pelvis width.100/Width of rib-arch)
v) Lower. tors width index B (Maximum width of thighs·100/Width of rib-arch)
vi) Lower-torso width index C (Maximum width of thighs·100/Pelvis width)
vii) Abdominal circumference index (Length of front abdomen·100/Minimum circumference of abdomen)
3) Lower Torso Formulae:
Each index was classified into five or six groups according to its numerical quantity, and each group was marked from one to six.
The Abdominal Circumference Index was designated as“U”, and the numerical formulae which were obtained as the result of arranging the markings in the order of their derivation: thusly, I II III A B C U, were called Lower Torso Formulae.
4) Only Lower Torso Index II and Lower Torso Width Index C derived from the mean numbers of each index were found to be absolutely equivalent when compared with the results of Dr. Shibata's observations of adult women in general residing in Tokyo.
In all other indices differences were noticed between the mean numbers, and except in the case of Lower Torso Index I, the arria were smaller.
However, when the shapes were fixed under Dr, Ogawara's classification system, the mean number of each index was“3”, resulting in exactly identical shapes.
5) There are remarkable differences in individual Lower Torso Formulae, but the Lower Torso Formulae obtained from the mean numbers of each index is 3333333; therefore, presenting the same as that of Shibata's for adult women in general.
6) In other 'words, absolutely no difference is seen in the Lower Torso Formulae between the ama and adult women in general, but compared to the length of their abdomen, the width of the upper part of the lower torso of the ama is wider than adult women in general and in comparing widths the upper part is better developed compared to the, lower part, while on the other hand, the circumference of the abdomen is bigger compared to the length of the abdomen.
These facts mean that the ama is wider at the upper part of the lower torso and has a bigger abdominal circumference than adult women in general.View full abstractDownload PDF (1141K) -
Shunji TOKUOKA1951 Volume 27 Issue 4 Pages 107-116,124
Published: July 20, 1951
Released on J-STAGE: September 24, 2012
JOURNAL FREE ACCESS1). Hemogram of 100 epileptics is almost always normal in the intervals of fits, but slight leucocytosis accompanying eosinophilia has been observed within several hours after grand mal seizures.
2). In 28 out of 33 idiopathic epileptics (84.8%) significant eosinopenia has been observed following caudal resection of the pancreas (3/5 of the entire organ), and persisted over 10 days in 18 cases (54.5%). The persisting postoperative eosinopenia has been more frequently observed in the cases, in which postoperative hyperglycemia was pronouced and longer lasted. The relation between alleviation of epileptic seizures and eosinopenia after the operation was not confirmed. In childhood the persisting postoperative eosinopenia has been rarely observed, but eosinophilia more frequently.
3). Following miscellaneous surgical operations, except caudal resection of the pancreas, significant eosinopenia has been observed in 83.3 per cent of 78 patients with or without epilepsy, but in all cases circulating eosinophils returned to or above preoperative levels within 4 days after operation.
4). Some relation between the number of circulating eosinophils, level of blood sugar, insulin and estrogen after caudal resection of the pancreas was found in epileptics. The persisting depression of circulating eosinophils following caudal resection of the pancreas in epileptics may be caused by an increase in adrenal activity. I suppose that the yet unknown mechanism, which regulates pituitary ACTH secretion, not based on the action of epinephrine, may be concerned with the change in metabolism of zinc following caudal resection of the pancreas.View full abstractDownload PDF (1870K) -
Kamon AKASHI, Norio AKASHI1951 Volume 27 Issue 4 Pages 117-120_1,125
Published: July 20, 1951
Released on J-STAGE: September 24, 2012
JOURNAL FREE ACCESS1. The authors have experienced 23 clinical examples of so-called stomach-hearts-symptomencomplex of Roemheld (1912), Lurje (1931) and Bergmann (1928).
2. This symptom seems to appear in comparatively high frequency in the younger group as attested by the fact that there were 15 cases, or 65.21%, in the 20 to 29 year age-group.
3. In all the symytomatic cases, instableness of the autonomous nervous system tending comparatively toward vagotonie was noticed.
There were distinct reactions in the atropin and pilocarpin tests.
An extreme case of stomach ptosis, caused stimulation of the pyloric region which was generally increased after over-eating and bathing.
As the result of X-ray inspections, stomach ptosis was observed in 19 examples or 82.60%of the cases. The beginning of this symptom can be proved when sulphuric barium passes through the pyloric region.
5. The principal symptoms are the same as that of temporary angina pectoris; namely, acute pain of the left chest, Beklemmung and intense pain of the left scapula. However, with the elvation of the abdominal region, change of physical position or vomition, there was relief from these symptoms or showed signs of such.
6. We believe this symptom has its beginning in the vegetative nerves which receive an oppressive stimulation from stomach ptosis within the hiatus oesophagus, resulting reflexively in the contraction of the coronary arteries.
Also hiatus hernia is believed to be caused due to the increased internal pressure of the peritoneal cavity, decreased internal pressure of the chest, relaxing of the diaphragm and compression of the oesophagus. And we are convinced that symptomencomplex is due to the instableness of the autonomous nervous system.
7. Injections of tetraeythylammmoniumbromide (T. E. A. B.) were comparatively effective for this symptom. (We used Newrogin 2 cc. manufactured by Tanabe Tokyo.)
In the five cases which T. E. A. B. was ineflective, we applied electroshock therapy, and we believe it is noteworthy that three of the cases were completely cured with a single treatment.
In regards to the general treatment of this symptom, particularly the relation between T. E. A.B. and electro-shock therapy, we are still continuing our experiments.View full abstractDownload PDF (1205K)
- |<
- <
- 1
- >
- >|