Folia Endocrinologica Japonica
Online ISSN : 2186-506X
Print ISSN : 0029-0661
ISSN-L : 0029-0661
Volume 24, Issue 1-4
Displaying 1-4 of 4 articles from this issue
  • 1948 Volume 24 Issue 1-4 Pages 1-6
    Published: September 20, 1948
    Released on J-STAGE: September 24, 2012
    JOURNAL FREE ACCESS
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  • [in Japanese]
    1948 Volume 24 Issue 1-4 Pages 7-9
    Published: September 20, 1948
    Released on J-STAGE: September 24, 2012
    JOURNAL FREE ACCESS
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  • Yoshio Tajima
    1948 Volume 24 Issue 1-4 Pages 10-17
    Published: September 20, 1948
    Released on J-STAGE: September 24, 2012
    JOURNAL FREE ACCESS
    Resection of both carotid sinus and aortic nerves do not produce the persistent arterial hypertension. As one of these causes, Sakurasawa and Saito habe pointed out the presense of pressoreceptoric nerve fibers in vagal trunks ('41), Further I have studied this subject about welltrained unnarkotised 3 dogs and 2 cats, measuring arterial blood pressure, heartrates, and respiratory rates during 2 years, After Eb. Koch's denervation of both carotid sinus, I resected the left cervical vagosympathetic trunk, and severed all cardiac branches beneath the emergence of right recurrentnerve, except for right vagal trunk. But I could gain neither permanent hypertonia nor hypertensive tachycardia in Mannaberg's sense; the respi-ratory rates also returned to normal range within very short time
    Moreover, I have observed the identical relationship in the manner of chemoreceptoric nerve, fibers (details in the sequel). At the last experiment, I have elucidated under the chloralose narcosis that both arterial pressure and cardiac rates reach to the adrenaline maximum by the resection of the remaining right vagal trunk. Autopsies were all examined and o regeneration was ascertained.
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  • Sigeji Kijima
    1948 Volume 24 Issue 1-4 Pages 18-24
    Published: September 20, 1948
    Released on J-STAGE: September 24, 2012
    JOURNAL FREE ACCESS
    A necrosis of the anterior pituitary following parturition in an autoptic case of puerpera was observed. In the literature 37 cases such as this necrosis due to pregnancy or parturition had been reported untill 1940 with various opinions on their causes. After the fine examination of clinical and patholoical findings of each case, it is concluded that such necrosis were an infarct caused by spasmodic contraction at the arteriole of the anterior pituitary as an anaphylactic shock brought by placental substances.
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