Folia Endocrinologica Japonica
Online ISSN : 2186-506X
Print ISSN : 0029-0661
ISSN-L : 0029-0661
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Volume 29, Issue 7-8
Displaying 1-4 of 4 articles from this issue
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  • [title in Japanese]
    [in Japanese]
    1953 Volume 29 Issue 7-8 Pages 155-188
    Published: November 20, 1953
    Released on J-STAGE: September 24, 2012
    DOIhttps://doi.org/10.1507/endocrine1927.29.7-8_155
    JOURNAL FREE ACCESS
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  • Effective Case of Transplantation of Caw-Pituitary and Adrenal Gland in Scleroderma
    Hajime YAMADA, Fumio OSAKA, Heiske HIGASHI
    1953 Volume 29 Issue 7-8 Pages 189-192,197
    Published: November 20, 1953
    Released on J-STAGE: September 24, 2012
    DOIhttps://doi.org/10.1507/endocrine1927.29.7-8_189
    JOURNAL FREE ACCESS
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    We met with a typical case of scleroderma generalisata, and found that dysfunction existed in the pituitary adrenal axis. Although injections of cortisone afforded only a temporary relief clinically, the transplantation of the cow-pituitary and the adrenal gland beneath the fascia lata bilaterally, improved both pituitary-adrenal function and subjective symptoms, especially the softening of the sclerosed skin and the alleviation of motorie disturbances were observed.
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  • Toxicity of Alloxan Measured with Heinz's Body
    (Research on Heinz's Body, 19th Report)
    Toru MIYAZAKI
    1953 Volume 29 Issue 7-8 Pages 193-196
    Published: November 20, 1953
    Released on J-STAGE: September 24, 2012
    DOIhttps://doi.org/10.1507/endocrine1927.29.7-8_193
    JOURNAL FREE ACCESS
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    Since the study of Alloxan diabetes by H. R. Jacobs (1937), researches on the experimental diabetes made a dramatic progress. In order to determine experimentally what kind of toxicity alloxan has upon blood elements, I measured Heinz's body.
    1) Analysis, by the in vitro formation method of Heinz's body
    2) Analysis, by the in vitro accelerated formation method of Heinz's body i) Experiment with normal saline with alloxan added.
    ii) Experiment with serum with alloxan added
    In above-mentioned conditions, Heinz's body shows a tendency to a slight increase only.
    For this result alone, I cannot declare with surety that alloxan is a remarkable blood toxin in so far as Heinz's body is concerned.
    If we assumed that alloxan might be a diabetogenic agent, we cannot explain solely with alloxan the phenomenon that in clinical diabetes a high acceleration of Heinz's body formation is found; other noxae than alloxan may be responsible for this phenomenon.
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  • [title in Japanese]
    [in Japanese]
    1953 Volume 29 Issue 7-8 Pages 198-216
    Published: November 20, 1953
    Released on J-STAGE: September 24, 2012
    DOIhttps://doi.org/10.1507/endocrine1927.29.7-8_198
    JOURNAL FREE ACCESS
    Download PDF (2933K)
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