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Volume 53, Issue 4
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[in Japanese]1962 Volume 53 Issue 4 Pages 265-290
Published: 1962
Released on J-STAGE: July 23, 2010
JOURNAL FREE ACCESSDownload PDF (3304K) -
PART 1: SOME BIOCHEMICAL STUDIES ON URINARY AND SERUM MUCOPROTEIN AND PROTECTIVE COLLOIDS IN URINE CAUSED BY TEMPORARY RENAL ISCLIEMIA IN RABBITS, AND ANOTHER HISTOCHEMICAL STUDIES ON THE KIDNEYHiroshi Moriwaki1962 Volume 53 Issue 4 Pages 291-307
Published: 1962
Released on J-STAGE: July 23, 2010
JOURNAL FREE ACCESSRenal calcification was experimentally induced in one side kidney by the means of temporal ischemia (for one and two hours) while another side of kidney was removed prior to the experiment and variations both of volumes and concentrations of urinary, serum mucoprotein and protective colloids in urine were investigated, and comparison of these results were made between changes of both urinary and of serum calcium levels. In addition, the histochemical study of kidney was also carried out (by the method of PAS and Von Kossa stain).
Following results were obtained:
1) High Levels of the mucoprotein excretion in urine were maintained during the oliguric period caused by renal ischemia. When urinary volume was recovered to the normal, the concentration and daily output of urinary mucopratein increased and lasted for 10 days: This was more prominent in the ischemic group of 2 hours.
2) Serum mucoprotein also showed the high level after ischemia in many cases. In these cases these values were parallel with urinary mucoprotein.
3) Although the values of protective colloids in urine increased after ischemia in the many cases, it was variable and any difinite relationship was not found between these values and those of urinary mucoprotein.
4) Urinary calcium slightly increased in early stage. Any significant change was not seen in serum calcium value.
5) Calcium deposits were seen in many cases of which renal blood supply was stopped for 2 hours. The main site of renal involvement was evident in renal tubules, and its necrotic lesion and casts were positive by PAS stain. Calcium deposits were developed over these lesions.
On these results it was considered that the most of urinary mucoprotein appeared to be the sum of those which was originated from serum mucoprotein and from the epithelium of degenerated renal tubules, and that the changes of this substance in renal tubules were very important as a cause of calcium deposits in ischemic kidney of rabbit.View full abstractDownload PDF (6012K) -
PART 2: SOME BIOCHEMICAL STUDIES BOTH ON URINARY AND SERUM MUCOPROTEIN AND ON PROTECTIVE COLLOIDS IN URINE DURING CALCIUM DEPOSITS IN RABBIT KIDNEY WITH ALLERGIC NEPHRITISHiroshi Moriwaki1962 Volume 53 Issue 4 Pages 308-323
Published: 1962
Released on J-STAGE: July 23, 2010
JOURNAL FREE ACCESSRenal shock was induced by injecting guinea pig serum sensitized by rabbit red cells into the renal artery of the rabbits, and histochemical changes in the kidney caused by this reversed anaphylaxis were found.
Mucoprotein in both urine and serum, protective colloids in urine and calcium values were measured.
In addition, histochemical studies of kidney were also carried out (by the method of PAS and Von Kossa stain).
1) The excretion of urinary mucoprotein evidenty increased in two or three days after the development of nephritis.
It was recovered to the control levels in 5th to 7th day with some variation. Serum mucoprotein also increased and showed correlation between urinary mucoprotein.
2) Protective colloids in urine also increased.
3) Any significant change in urinary and serum calcium levels were not seen.
4) Histochemical section of the kidney showed the picture similer to acute localized nephritis, but there were few changes in renal tubules.
Gromerulus, tubular epithelium and tubular casts were positive by PAS stain. This appearance may be due to the following conditions: (1) the increased permiability of blood vessels, (2) the degeneration of tubules, (3) the changes of reabsorption capacity of tubules caused by allergic nephritis.
Relation between renal calcification and allergic nephritis was discussed, and emphasis was made that alteration of urinary and serum mucoprotein in allergic nephritis may be considered as a cause of calcification in kidney.View full abstractDownload PDF (5653K) -
Tokuji Ichikawa, Kazumoto Ito, Etsuji Takasaki, Tsutomu Goro1962 Volume 53 Issue 4 Pages 324-334
Published: 1962
Released on J-STAGE: July 23, 2010
JOURNAL FREE ACCESSDownload PDF (13550K) -
Takeshi Minami, Ken Koshiba, Akiyo Taguchi1962 Volume 53 Issue 4 Pages 335-343
Published: 1962
Released on J-STAGE: July 23, 2010
JOURNAL FREE ACCESSUrinary tract infection, frequently troublesome and difficult to eradicate, has been considered an almost inevitable occurrence following transurethral prostatic resection. In an effort to treat urinary infection after transurethral prostatic resection, we have used trypure, a proteolytic enzyme, in 14 cases.
Nine patients were treated by bladder irrigation with 50cc of 20mg/dl trypure normal saline solution for 6 times in average, and bacteriological cure were obtained in 2 and improvement in 4 patients. The rest of the 5 patients were treated with 40mg/dl trypure normal saline solution in the same manner as above, and bacteriological cure were obtained in 1 and improvement in 3 patients. In total, 11 of the 14 patients (78.6%) were successfully treated by this treatment.
No unpleasant side effect occurred in our cases.
We feel that this treatment is an effective and recommendable therapeutic procedure for combating urinary tract infection following transurethral prostatic resection.View full abstractDownload PDF (1362K)
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