The Japanese Journal of Urology
Online ISSN : 1884-7110
Print ISSN : 0021-5287
Volume 50, Issue 11
Displaying 1-8 of 8 articles from this issue
  • Kojiro Baba
    1959 Volume 50 Issue 11 Pages 1113-1167
    Published: November 20, 1959
    Released on J-STAGE: July 23, 2010
    JOURNAL FREE ACCESS
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  • PART 1. THE BLOOD LEVELS IN HYPERPLASIA OF PROSTATE
    Mitsugu Kamidate
    1959 Volume 50 Issue 11 Pages 1168-1178
    Published: November 20, 1959
    Released on J-STAGE: July 23, 2010
    JOURNAL FREE ACCESS
    Concerning the investigation of the neoplasms of prostate, the author reported on the blood dextrose levels in prostatic hyperplasia and the results were as followed.
    1. On the blood dextrose level in prostatic hyperplasia, 85.6mg/dl was obtained with 13 patients as preoperative levels. No valuable differences could be found between preoperative levels.
    2. On the glucose tolerance test with intravenous injection of 50% glucose, 15 cases of 20 cases (70%) of prostatic hyperplasia showed the decreases of levels. 3 cases of 10 cases (30%) showed much improvements after the prostatectomy.
    3. On the causes of low levels of glucose tolerance, it depends not only on the disturbance of urination but also on the disturbance of internal secretion, and there is some intimate relationship with slight hepatic disturbance.
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  • PART 2. THE BLOOD LEVELS IN PROSTATIC NEOPLASMS
    Mitsugu Kamidate
    1959 Volume 50 Issue 11 Pages 1179-1182
    Published: November 20, 1959
    Released on J-STAGE: July 23, 2010
    JOURNAL FREE ACCESS
    On the blood dextrose levels in prostatic hyperplasia, the author has reported already in part 1. In this studies, the same experiments have been performed on prostatic neoplasms and the results were as followed.
    1. The blood dextrose level in prostatic neoplasms was 85.0mg/dl with 7 patients.
    2. 6 cases out of 7 cases (85.7%) showed the decreases of the blood levels with the glucose tolerance tests and no distinct differences could be found between hyperplasia and neoplasms of prostate gland.
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  • PART 3. THE EFFECTS OF THE PROSTATIC EXTRACTS ON CARBOHYDRATE METABOLISM IN RABBITS
    Mitsugu Kamidate
    1959 Volume 50 Issue 11 Pages 1183-1190
    Published: November 20, 1959
    Released on J-STAGE: July 23, 2010
    JOURNAL FREE ACCESS
    The effects of the prostatic extracts of 10% saline, 1% polysaccharide and 1% polypeptide on carbohydrate metabolism in rabbits were reported and the results were as followed.
    1. The slight increase of the blood dextrose levels could be found with the administration of these extracts respectively.
    2. The glucose tolerance curves in normal male rabbits showed no definite results by using 50% glucose, but each curve in each animal showed the definite result within 10mg/dl differences. By the continuous administration of 1% polypeptide extract for 10 days, the results of the glucose tolerance test showed a slight decrease and the tolerance curve showed slight extension comparing with normal curve.
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  • THE EFFECTS OF MALE SEX HORMONES ON BLOOD DEXTROSE LEVELS
    Mitsugu Kamidate
    1959 Volume 50 Issue 11 Pages 1191-1194
    Published: November 20, 1959
    Released on J-STAGE: July 23, 2010
    JOURNAL FREE ACCESS
    Concerning the physiology of prostate gland, the internal secretion, especially, androgen have strong influences upon the prostate gland. The author experienced the effects of testosterone propionate (30mg/day for 10days) on blood dextrose level in male adult rabbits and castrated rabbits. The blood levels showed normal and could not found any differences between normal and castrated rabbits.
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  • Takanori Kameda
    1959 Volume 50 Issue 11 Pages 1195-1206
    Published: November 20, 1959
    Released on J-STAGE: July 23, 2010
    JOURNAL FREE ACCESS
    Ascorbic acid in blood and urine was determined at pre- and post-treatment (prostatectomy and hormone therapy) by dinitrophenylhydrazine method on 15 patients with prostatic tumor. The experiments were carried out by ascorbic acid tolerance test with 500mg of ascorbic acid.
    1. Ascorbic acid, in general, at preoperation showed low level and much differences could not be found between prostatic hypertrophy and tumor by the mean value in blood and urine.
    2. Much improvements could be found in ascorbic acid level at postoperation (3-4 weeks later) in prostatic hypertrophy, and the more supplies of ascorbic acid at postoperation showed the better improvements.
    3. On the contrary, in prostatic tumor, no improvements could be found at postoperation. The excretion of ascorbic acid in urine was rather diminished and it would be expected to have a stimulating effect on metabolism.
    4. No improvements could be found by the estrogen treatment and Co60 therapy. These treatment would be suggested to have some effects on ascorbic acid metabolism.
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  • PART 6. STUDIES ON PROSTATIC EXTRACTS
    Takanori Kameda
    1959 Volume 50 Issue 11 Pages 1207-1213
    Published: November 20, 1959
    Released on J-STAGE: July 23, 2010
    JOURNAL FREE ACCESS
    Ascorbic acid in the pituitary body, adrenal gland, spleen, liver, kidney, testis, prostatic gland and in blood was determined by the administration of prostatic extracts for 10 days. The experiments were carried out on 3, 7, 10, 14, 21 days after the administration of prostatic extracts.
    Ascorbic acid in most organs except testis showed obvious decreases, especially, remarkable decreases could be found in adrenal, liver, kidney and prostatic gland.
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  • Takanori Kameda
    1959 Volume 50 Issue 11 Pages 1214-1224
    Published: November 20, 1959
    Released on J-STAGE: July 23, 2010
    JOURNAL FREE ACCESS
    Ascorbic acid in the pituitary body, adrenal gland, spleen, liver, kidney, testis, prostatic gland and in blood was determined by the administration of ACTH, cortisone, adrenalin and the effects of these hormones were investigated.
    Ascorbic acid in blood diminished remarkably by adrenalin, indefinite by cortisone and not always increased by ACTH.
    Ascorbic acid in adrenal gland showed remarkable decrease by ACTH and slightly decrease by adrenalin.
    Ascorbic acid in spleen showed slightly decrease by ACTH and, contrarily, moderate increase in liver.
    In testis, special changes could not be found and also, no significant changes could be found in prostatic gland. The physiological response of ascorbic acid on male sexual accessary organs could not be solved.
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